Kamikaze1414779018

i was there, and it was fantastic, just chill, then bimb after bomb

but the Techno will still be Playing !

hey Ramom, How about we do this in Detroit ! I think it will OWN here ! get a hold of me !

Physical examination Many patients do not voluntarily report snoring to their physicians because of the social embarrassment often associated with this problem. The author has found the simple question, "Has anybody mentioned to you that you snore from time to time?" to be extremely helpful and nonconfrontational. The author has found several structural predictors of OSA to be useful. Most patients with sleep apnea are overweight and have short, thick necks. Increasing neck circumference is linearly related to the probability of OSA and may be more specific than body mass index (BMI) in the clinical diagnosis of OSA. Maxillary and mandibular deficiency is an important finding and can be initially examined by evaluating the dental occlusion. The absence of mandibular teeth may also lead to mandibular atrophy. Examination of the oropharynx often reveals an elongated uvula, a small oropharyngeal opening, a large tongue, and prominent oropharyngeal folds. The uvula may telescope upon itself when the patient says "ah," indicating an increased possibility that OSA is present. A recent study demonstrated a 70% positive predictive value of tongue scalloping and OSA. Scalloping of the tongue was defined as the multiple lateral glossal indentions that result from molar compression (Weiss, 2005). Occasionally, large tonsils are seen in adults, but this is found more often in pediatric patients. Direct fiberoptic examination or indirect mirror examination may reveal a mass or tumor in the upper airway or possible deviation of the nasal septum. The author always performs a complete upper airway examination to exclude unusual causes for upper airway obstruction, such as a neoplasm. Completely examine the nose, nasopharynx, oral cavity, oropharynx, hypopharynx, larynx, and neck. In the Müeller maneuver, the patient inhales with their nasal passages occluded and their lips closed while the airway is examined with a flexible fiberoptic laryngoscope. Ascertaining the level of greatest obstruction is often helpful but can be difficult, particularly in the pharynx. The findings of a Müeller maneuver, for example, dramatically differ from the sleep-breathing situation. In the surgical candidate, video sleep nasoendoscopy (VSE) has been advocated to assess the sites of obstruction. Close attention should be paid to the levels of the soft palate, lateral pharyngeal wall, tonsils, tongue base, epiglottis, and hypopharynx (the piriform fossae that collapses in around the larynx; Abdullah, 2003). This technique has been criticized in the past for inducing false-positive results (ie, causing snoring and obstruction in a patient who normally does not have this problem). A recent study showed that sleep nasendoscopy can be used without falsely producing snoring and obstruction in patients if propofol and a pump with a microprocessor that allows a controlled, determined intravenous infusion rate are used (Berry, 2005).

Pathophysiology Three factors involved in the development of OSA include decreased dilating forces of the pharyngeal dilators, upper-airway anatomic abnormalities, and the negative inspiratory pressure the diaphragm generates. The site of obstruction is primarily in the pharynx; however, many anatomic sites clearly contribute. The muscles of the upper airway, including the sternohyoid, genioglossus, and tensor palatini, work together to dilate or stiffen the extrathoracic airway and to maintain its airway caliber. Collapse may begin when the base of the tongue abuts the posterior pharyngeal wall and soft palate. This may progress to the lower pharynx. The exact cause of upper airway collapse in humans has not been completely elucidated. An animal study, however, revealed nearly abolished genioglossal activity during rapid eye movement (REM) sleep, even in the presence of elevated inspired carbon dioxide levels (Parisi, 1988). Data from recent studies have also suggested that nasal obstruction plays a prominent role in OSA. Extended or excessive tissue of the soft palate, a large tongue base, a large uvula, large tonsils, and redundant pharyngeal mucosa are correlated with a narrowed upper airway. With airway narrowing, increased inspiratory pressure is needed to maintain adequate ventilation. A virtual vacuum on inspiration promotes further collapse of the soft tissue, which often has poor tone due to repeated vibratory trauma. Of importance is the finding that increased pulmonary resistance also requires increased negative inspiratory pressures. Nocturnal oxygen desaturation and hypercapnia associated with OSA increases arterial blood pressure in both the systemic and pulmonary circulations. Over time, hypertension can lead to cardiac hypertrophy and decompensation. Cor pulmonale is a classic clinical manifestation of long-standing OSA. The Sleep Heart Study and Wisconsin Sleep Cohort Studies provide the most compelling evidence that patients with sleep-disordered breathing have a significantly greater risk of developing hypertension and requiring antihypertensive medications (Nieto, 2000; Peppard, 2000). Arrhythmias can also occur as a result of cardiopulmonary strain secondary to hypoxia. In rare cases, this may lead to nocturnal death. Diminished oxygen saturation also stimulates erythropoiesis and clinical polycythemia. Additionally, OSA has been implicated as a risk factor for first stroke, recurrent stroke, and poststroke mortality (Dyken, 1996). Clinical History A useful source for obtaining a history for a patient who snores is the patient's bed partner. Typical symptoms include snoring, apneic episodes (witnessed by a bed partner), excessive daytime somnolence, and difficulty with memory and cognition. Other indicators might include enuresis (bed wetting) or a history of maxillofacial trauma. Patients who are referred for a surgical evaluation often report failed treatment with continuous positive airway pressure (CPAP).

hey Mirza, Whats up man? its been forever I am in healthcare and deal with this every day as I do medical sales now and help the physicians prescribe the above mentioned "sleep machine Please look into the terms Sleep Apnea, Sleep disordered breathing, CPAP, BiPAP I do sell CPAP / BiPAP which is the most common and effective treatment for Sleep Apnea , for http://www.lincare.com/ IF you have any questions, please feel free to PM me and I can talk to you more in person about this. ---Eric Kamikaze and read on Snoring, excessive daytime somnolence, restless sleep, and apnea are manifestations of sleep-disordered breathing, which has plagued society for centuries. Recent understanding of the pathophysiology related to these problems has led to some successes in both nonsurgical and surgical interventions. Numerous sleep disorders are organized in the International Classification of Sleep Disorders by the American Sleep Disorders Association. The primary disorders that may warrant surgical intervention include snoring and obstructive sleep apnea (OSA). The otolaryngologist's approach to management and treatment of these conditions is discussed below. Problem Snoring is an undesirable sound that originates from the soft tissues of the upper airway during sleep. It is usually a source of contention for patients and their bed or dwelling partners, and it may be a harbinger of something more serious, such as OSA. OSA is a sleep disorder in which airflow is repeatedly reduced or ceased. The disorder may vary in severity and is often associated with other physiologic problems. These problems include altered mood and behavior (depression, lethargy, cognitive and memory impairment), morning headaches, decreased libido, systemic and pulmonary hypertension, congestive heart failure, and sleep-related arrhythmias, among others. Apnea is obstructive only when polysomnography reveals a continued inspiratory effort evidenced by abdominal and thoracic muscle contraction. In central apnea, absence of airflow accompanies a lack of inspiratory effort, and this condition is not amenable to surgical correction. At times, apnea may be mixed, occurring with both obstructive and central apnea symptoms. Patients with this condition present a therapeutic challenge to the surgeon. Frequency The exact prevalence of OSA is unknown, but most experts agree it is frequently undiagnosed. A large study of 602 patients showed that 28% of women and 44% of men aged 30-60 years reported habitual snoring. Polysomnography demonstrated that 9% of women and 24% of men had a respiratory disturbance index (RDI) of 5 or higher, suggesting some degree of sleep apnea. Age: OSA can occur at any age, but it is most commonly diagnosed in patients aged 45-65 years. Sex: In adults, the male-to-female ratio is approximately 2:1. Etiology Snoring is a result of incomplete pharyngeal obstruction. Turbulent airflow and subsequent progressive vibratory trauma to the soft tissues of the upper airway are important factors that contribute to the condition. Anatomic obstruction leads to increased negative inspiratory pressure, which propagates further airway collapse, turbulence, and noise. The imbalance between the forces that act to maintain airway patency (the force of the pharyngeal muscles) and the negative inspiratory forces generated by the diaphragm is thought to be the primary etiology of anatomic obstruction in OSA. In OSA, the tongue contacts the soft palate and posterior pharyngeal wall in the presence of lateral collapse of the pharynx, generating occlusion. Significant factors that contribute to this condition include obesity, redundant tissue in the neck, retrognathia, and craniofacial anomalies. In addition, anatomic abnormalities of the nasal airway (eg, septal deviation, inferior turbinate hypertrophy, nasal-valve narrowing, adenoid hypertrophy) may play a role. Alcohol and other sedatives may increase the severity of OSA. Data from a recent meta-analysis by Rada also suggested a causal relationship between OSA and head and neck cancer (which may first manifest as OSA).

the above photo is nice, with a large amount of Photoshop work put into it, the guy most likely did a huge amount of Sharpening on the entire photo to get the detail to pop, and like POD said then used HDR. One of the few landscape photos I like that I havr taken is this below. It is a good photo and pretty much straight from the camera with very little photos hop done to it. I might be able to go and redo this shop soon and apply the same skills as sharpening and HDR if I get a time.

they were also just here Memorial day weekend at DEMF, but now they will play longer and harder for a full set ! This will be nuts and a can't miss show for all Techno freaks !

...Saturday September 1st, 2007 Paxahau 9 Year Anniversary... <<<featuring>>> Hardfloor - live - Germany Hardfloor played in Detroit for the first time in their 16 year career at this years Movement Festival in May. Their show was arguably one of the best of the weekend, whipping all of Hart Plaza into a frenzy as they played through a decades' worth of their highly successful anthems. Their show in a more intimate atmosphere promises to be a rare club experience that you do not want to miss. Stacey Pullen John Arnold Matthew Dear Ryan Elliott Eric Johnston Mike Servito Keith Kemp Matt Clarke Drew Maddox Shawn Michaels Vacuum (aka Matt Abbott) Anco Tozy (aka Andy Toth & Colin Z.) - live Pax.iPod 2pm - 10pm / Hoot's on the Avenue - 18 & up indoor / outdoor venue - 1460 Michigan - Detroit 10pm - 2am / Bleu Nightclub - 18 & up downstairs open - 1540 Woodward - Detroit Presale tickets available via paxahau.com / Recordtime Ferndale Limited amount of early bird $5 & $10 tickets available... **complimentary shuttle venue to venue will run from 10-2:30am**

DDDDDDDDDEEEEEEEETROIT !

When the F are they gonna come out with info? I've been waiting for a while now. Inquiring minds want to know. They told me about 6 weeks before there should be some info. They may be working on something huuuuuge! << deeeeeeeeeeeeeeeeeeeeeetroit Book your flights NOW !

can someone convert this into a regular DL and not a podcast , Remon help

Heater @ DEMF 9GnuAbShkMk